I thought the point was that the double bonded agents stick together and are just excreted and the bonds don't easily get broken at all.

I thought the problem was with older agents there were single bonds that could be broken in the chain and that's what can cause the build-up.

But I was under the impression those were phased out over a decade ago.

So is this saying even the strong double-bonded ones are somehow building up in some way we don't understand?

It's also been known forever that these agents are riskier in patients with kidney failure, and that's directly factored into the algorithms doctors use and has been forever.

So.... what's the point of this? Is it rage-bait?